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Tissue inhibitor of metalloproteinases-1 protects the human breast carcinoma cell line MCF-7 S1 against anthracycline-induced cell death by activation of the akt survival pathway
British Library Conference Proceedings | 2009| -
Cathepsin B Mediates Tumor Necrosis Factor-induced Arachidonic Acid Release in Tumor Cells
British Library Online Contents | 2002| -
TIMP-1 overexpression does not affect sensitivity to HER2-targeting drugs in the HER2-gene-amplified SK-BR-3 human breast cancer cell line
British Library Online Contents | 2013| -
A TIMP-1 splice variant transcript: Possible role in regulation of TIMP-1 expression
British Library Online Contents | 2008| -
Diarylurea Compounds Inhibit Caspase Activation by Preventing the Formation of the Active 700-Kilodalton Apoptosome Complex
British Library Online Contents | 2003| -
Identification of alternatively spliced TIMP‐1 mRNA in cancer cell lines and colon cancer tissue
Free accessWiley | 2007| -
Characterization of Anti-TIMP-1 Monoclonal Antibodies for Immunohistochemical Localization in Formalin-fixed, Paraffin-embedded Tissue
British Library Online Contents | 2006| -
TIMP1 overexpression mediates resistance of MCF-7 human breast cancer cells to fulvestrant and down-regulates progesterone receptor expression
British Library Online Contents | 2013| -
Cathepsin B acts as a dominant execution protease in tumor cell apoptosis induced by tumor necrosis factor
British Library Online Contents | 2001| -
Investigating the biomarker potential of glycoproteins using comparative glycoprofiling — application to tissue inhibitor of metalloproteinases-1
British Library Online Contents | 2008| -
Establishment and Characterization of 7 New Monoclonal Antibodies to Tissue Inhibitor of Metalloproteinases-1
British Library Online Contents | 2005| -
Identifying sources and estimating glandular output of salivary TIMP‐1
Taylor & Francis Verlag | 2008| -
Regular Articles - Cathepsin B acts as a dominant execution protease in tumor cell apoptosis induced by tumor necrosis factor.
Online Contents | 2001|Contributors: Lademann, U.
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